Static ataxia
In the former the ataxia is not influenced by closing the eyes; in the latter (the sensory impulses going up the posterior columns and medial fillet to the thalamus and post central cortex) shutting out visual afferent impulses going to the brain aggravates or brings out the ataxia. From a clinical study or postural body reflexes, I have concluded that the Rom berg sign may on occasion be interpreted as a loss of optic righting reflexes. (Read on …)
Equilibrator Coordination
Walking backward is much easier in paresis or paralysis of the quadriceps femora’s then walking forward. To elicit further the presence of equilibrator ataxia in walking the following tests are employed: the patient is asked to rise on command and thirdly, he is asked to walk and turn around quickly ob sharp command. The ataxic gait is thereby brought out even though it be absent in normal walking.
Normally there is associated swinging of the arms in walking. These movements are quite automatic and they are lost in paralysis agates (Read on …)
Gait
The gait may be spastic, stiff, with the legs extended, the feet shuffling. It may be hemiplegic that is the lower extremity is held stiff and circumfused in walking. In bilaterally spastic limbs as in cerebral diploid or ordinary paraplegia there is cross progression or scissor gait. The patient may take very short steps. The gait may be unsteady, the step irregular, jerky ataxic, the ledge being flung aimlessly or lifted unduly high from the ground. The patient watches his step in tab tic ataxia. He may be very unsteady, drunken, (Read on …)
« Previous Page